Gehirne von Alzheimerpatienten zeigen viele Veränderungen. Ablagerungen, schlechte Durchblutung, Nervenzellen, die von innen verwachsen sind. Die Gründe hierfür sind ebenfalls vielfältig, Kohlenhydrate, die unkontrolliert mit Eiweißen reagieren, Probleme mit dem Reinigungssystem des Gehirns und auch die fehlende Nutzung.
Chronic stress, sleep deficits, and mood disturbance, conditions common in those with cognitive impairment, have been prospectively linked to the development and progression of both chronic illness and memory loss and are significant predictors of AD.
Although the mechanisms underlying the putative beneficial effects of meditation on cognitive, psychological, and physical health are not yet well understood, the observed changes likely occur through at least four pathways (41, 46, 84, 85, 101, 119). First, by reducing activation and reactivity of the sympathoadrenal system and the HPA axis and promoting feelings of well-being, meditation may alleviate the effects of stress, enhance sleep and mood, and foster multiple positive downstream effects on cognition, neuroendocrine status, neurological and metabolic function and related inflammatory responses (Figure 1, pathway 1). Second, meditation may enhance parasympathetic output, possibly via direct vagal stimulation, and thereby shift the autonomic nervous system balance from primarily sympathetic to parasympathetic, leading to positive changes in cardiovagal function, in mood, sleep, and energy state, and in related neuroendocrine, metabolic, and inflammatory responses, in turn, reducing risk for depression and cognitive decline (Figure 1, pathway 2). Third, findings of recent neuroimaging and neurophysiological studies (98, 106, 120) suggest that meditation, by selectively activating specific neurochemical systems and brain structures associated with positive mood, attention, and memory, may likewise promote beneficial changes in sympathetic/parasympathetic balance, in neurological structure and function, in affect and memory, and in related metabolic and inflammatory responses (Figure 1, pathway 3). Finally, findings of a recent study in dementia caregivers (121) suggest that meditation may also, by directly or indirectly stimulating increased telomerase activity, help promote telomere maintenance and buffer the effects of stress-induced cellular aging, thereby helping to preserve immune function and possibly reduce neuronal loss and other degenerative changes associated with aging and cognitive decline (122, 123). As discussed below, reductions in telomerase activity and telomere length have been linked to stress, depression, sleep loss, and cognitive impairment (99, 124–135) and shown to predict cognitive decline in both clinical and non-clinical populations (136, 137). Likewise, recent research in healthy adults (138–140), lonely older adults (141), and depressed dementia caregivers (142, 143) suggest that meditation may also buffer or reverse multiple stress-related changes in specific gene expression pathways implicated in the development and progression of AD, including those regulating oxidative stress, inflammation, cellular aging, and other factors contributing to impaired brain structure and function, and ultimately, to cognitive decline
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Quelle: Innes KE, Selfe TK. Meditation as a therapeutic intervention for adults at risk for Alzheimer's disease - potential benefits and underlying mechanisms. Front Psychiatry. 2014;5:40.
© 2026 Dr. Ulrich Strunz
